Atm haploinsufficiency results in increased sensitivity to sublethal doses of ionizing radiation in mice
- 1 April 1999
- journal article
- research article
- Published by Springer Nature in Nature Genetics
- Vol. 21 (4) , 359-360
- https://doi.org/10.1038/7684
Abstract
No abstract availableKeywords
This publication has 11 references indexed in Scilit:
- The murine gene p27Kip1 is haplo-insufficient for tumour suppressionNature, 1998
- Transforming growth factor-β1 is a new form of tumor suppressor with true haploid insufficiencyNature Medicine, 1998
- ATAXIA-TELANGIECTASIA AND THE NIJMEGEN BREAKAGE SYNDROME: Related Disorders But Genes ApartAnnual Review of Genetics, 1997
- Heterozygous ATM mutations do not contribute to early onset of breast cancerNature Genetics, 1997
- AT-tributable risks?Nature Genetics, 1997
- Molecular genotyping shows that ataxia-telangiectasia heterozygotes are predisposed to breast cancerCancer Genetics and Cytogenetics, 1996
- Pleiotropic defects in ataxia-telangiectasia protein-deficient miceProceedings of the National Academy of Sciences, 1996
- Targeted disruption of ATM leads to growth retardation, chromosomal fragmentation during meiosis, immune defects, and thymic lymphoma.Genes & Development, 1996
- Dual roles of ATM in the cellular response to radiation and in cell growth control.Genes & Development, 1996
- Atm-Deficient Mice: A Paradigm of Ataxia TelangiectasiaCell, 1996