Peptide Inhibition of ENaC
- 9 December 1998
- journal article
- research article
- Published by American Chemical Society (ACS) in Biochemistry
- Vol. 38 (1) , 354-363
- https://doi.org/10.1021/bi981979s
Abstract
No abstract availableKeywords
This publication has 20 references indexed in Scilit:
- Subunit Stoichiometry of a Core Conduction Element in a Cloned Epithelial Amiloride-Sensitive Na+ ChannelBiophysical Journal, 1998
- Mutations causing Liddle syndrome reduce sodium-dependent downregulation of the epithelial sodium channel in the Xenopus oocyte expression system.Journal of Clinical Investigation, 1998
- Liddle syndrome: An autosomal dominant form of human hypertensionKidney International, 1998
- Genotype–phenotype analysis of a newly discovered family with Liddleʼs syndromeJournal Of Hypertension, 1997
- Liddle disease caused by a missense mutation of beta subunit of the epithelial sodium channel gene.Journal of Clinical Investigation, 1996
- Triple-barrel Organization of ENaC, a Cloned Epithelial Na+ ChannelPublished by Elsevier ,1996
- Hypertension caused by a truncated epithelial sodium channel γ subunit: genetic heterogeneity of Liddle syndromeNature Genetics, 1995
- Liddle's syndrome: A public health menace?American Journal of Kidney Diseases, 1995
- Liddle's syndrome, an underrecognized entity: A report of four cases, including the first report in black individualsAmerican Journal of Kidney Diseases, 1995
- Liddle's Syndrome Revisited -- A Disorder of Sodium Reabsorption in the Distal TubuleNew England Journal of Medicine, 1994