DIFFERENCES IN ACTIVE-SITE STRUCTURE IN A FAMILY OF BETA-GLUCAN ENDOHYDROLASES DEDUCED FROM THE KINETICS OF INACTIVATION BY EPOXYALKYL BETA-OLIGOGLUCOSIDES
- 25 March 1989
- journal article
- research article
- Vol. 264 (9) , 4939-4947
Abstract
The active sites of a spectrum of .beta.-glucan endohydrolases with distinct, but related substrate specificities have been probed using a series of epoxyalkyl .beta.-glycosides of glucose, cellobiose, cellotriose, laminaribiose, laminaritriose, 3O-.beta.-D-glucosyl-cellobiose and 4O-.beta.-D-glucosyl-laminaribiose with different aglycon chain lengths. The inactivation of each of the endohydrolases by these compounds results from active site-directed inhibitor action, as indicated by the dependence of the inactivation rate on pH, glycosyl chain length and linkage position, aglycon length, and the protective effect of disaccharides derived from the natural substrates. Comparison of inhibitor specificity between a Bacillus subtilis 1,3;1,4-.beta.-D-glucan 4-glucanohydrolase (EC 3.2.1.73), a Streptomyces cellulase (EC 3.2.1.4), a Schizophyllum commune cellulase (EC 3.2.1.4), a Rhizopus arrhizus 1,3-(1,3;1,4)-.beta.-D-glucan 3(4)-glucanohydrolase (EC 3.2.1.6), and a Nicotiana glutinosa 1,3-.beta.-D-glucan 3-glucanohydrolase (EC 3.2.1.39) demonstrated different tolerances for glycosyl linkage positions in the inactivation process and a critical role of aglycon length reflecting differences in the active site geometry of the enzymes. For the B. subtilis endohydrolase it was concluded that the aglycon residue of the inhibitor spans the glycosyl binding subsite occupied by the 3-substituted glucosyl residue involved in the glucosidic linkage cleaved in the natural substrate. Appropriate positioning of the inhibitor epoxide group with respect to the catalytic amino acids in the active site is crucial to the inactivation step and the number of glucosyl residues in the inhibitor affects aglycon chain length specificity. The importance of this effect differs between the glucanases tested and may be related to the number of glycosyl binding subsites in the active site.This publication has 22 references indexed in Scilit:
- Inhibition of formation of complex oligosaccharides by the glucosidase inhibitor bromoconduritol.Proceedings of the National Academy of Sciences, 1982
- Isolation and structure of a tryptic glycopeptide from the active site of β-glucosidase A3 from Aspergillus wentiiBiochimica et Biophysica Acta (BBA) - Protein Structure, 1980
- Affinity labeling of creatine kinase by N-(2,3-epoxypropyl)-N-amidinoglycine.Journal of Biological Chemistry, 1979
- Amino acid sequence at the active site of β-glucosidase a from bitter almondsBiochimica et Biophysica Acta (BBA) - Enzymology, 1978
- Partial amino acid sequences around the essential carboxylate in the active sites of the intestinal sucrase-isomaltase complex.Journal of Biological Chemistry, 1976
- PYRUVATE CARBOXYLASE .V. INTERACTION OF ENZYME WITH ADENOSINE TRIPHOSPHATE1965
- Inactivation of Myosin by 2,4-Dinitrophenol and Protection by Adenosine Triphosphate and Other Phosphate CompoundsJournal of Biological Chemistry, 1963
- Esters of Methanesulfonic Acid as Irreversible Inhibitors of AcetylcholinesteraseJournal of Biological Chemistry, 1962
- ENZYMATIC PROPERTIES OF A BETA-GLUCANASE FROM BACILLUS SUBTILIS1961
- PROTEIN MEASUREMENT WITH THE FOLIN PHENOL REAGENTJournal of Biological Chemistry, 1951