Expression of bcl-2, bcl-x, bax and bak in Renal Parenchyma, Oncocytomas and Renal Cell Carcinomas
- 31 December 1998
- journal article
- Published by Elsevier in Pathology - Research and Practice
- Vol. 194 (12) , 837-845
- https://doi.org/10.1016/s0344-0338(98)80086-1
Abstract
No abstract availableKeywords
This publication has 18 references indexed in Scilit:
- Conversion of Bcl-2 to a Bax-like Death Effector by CaspasesScience, 1997
- Expression of the cell death-inducing gene bax in carcinomas developed from the follicular cells of the thyroid glandJournal of Clinical Endocrinology & Metabolism, 1996
- Induction of apoptosis by the Bcl-2 homologue BakNature, 1995
- Cloning of a bcl-2 homologue by interaction with adenovirus E1B 19KNature, 1995
- Apoptosis and expression of the bcl-2 proto-oncogene in the fetal and adult human kidney: Evidence for the contribution of bcl-2 expression to renal carcinogenesisHuman Pathology, 1994
- Identification of immunosuppressant-induced apoptosis in a murine B-cell line and its prevention by bcl-x but not bcl-2.Proceedings of the National Academy of Sciences, 1994
- The prevalence of BCL-2 immunoreactivity in breast carcinomas and its clinicopathological correlates, with particular reference to oestrogen receptor statusVirchows Archiv, 1994
- bcl-x, a bcl-2-related gene that functions as a dominant regulator of apoptotic cell deathCell, 1993
- BCL2 protein is topographically restricted in tissues characterized by apoptotic cell death.Proceedings of the National Academy of Sciences, 1991
- Prognostic significance of morphologic parameters in renal cell carcinomaThe American Journal of Surgical Pathology, 1982