Expression of the 1918 Influenza A Virus PB1-F2 Enhances the Pathogenesis of Viral and Secondary Bacterial Pneumonia
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- 1 October 2007
- journal article
- research article
- Published by Elsevier in Cell Host & Microbe
- Vol. 2 (4) , 240-249
- https://doi.org/10.1016/j.chom.2007.09.001
Abstract
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This publication has 35 references indexed in Scilit:
- Replication fitness determines high virulence of influenza A virus in mice carrying functional Mx1 resistance geneProceedings of the National Academy of Sciences, 2007
- Genomic analysis of increased host immune and cell death responses induced by 1918 influenza virusNature, 2006
- Influenza A Virus PB1-F2 Protein Contributes to Viral Pathogenesis in MiceJournal of Virology, 2006
- Insights into the Interaction between Influenza Virus and PneumococcusClinical Microbiology Reviews, 2006
- Bacterial Sinusitis and Otitis Media following Influenza Virus Infection in FerretsInfection and Immunity, 2006
- Influenza Virus PB1-F2 Protein Induces Cell Death through Mitochondrial ANT3 and VDAC1PLoS Pathogens, 2005
- Influenza Virus Neuraminidase Contributes to Secondary Bacterial PneumoniaThe Journal of Infectious Diseases, 2005
- Influenza-Associated Hospitalizations in the United StatesJAMA, 2004
- The Influenza A Virus PB1-F2 Protein Targets the Inner Mitochondrial Membrane via a Predicted Basic Amphipathic Helix That Disrupts Mitochondrial FunctionJournal of Virology, 2003
- Observations ON INFLUENZA AND ITS COMPLICATIONSBMJ, 1919