Trimetazidine Counteracts the Hepatic Injury Associated with Ischemia-Reperfusion by Preserving Mitochondrial Function
- 1 July 1998
- journal article
- Published by Elsevier in The Journal of Pharmacology and Experimental Therapeutics
- Vol. 286 (1) , 23-28
- https://doi.org/10.1016/s0022-3565(24)37552-4
Abstract
No abstract availableThis publication has 17 references indexed in Scilit:
- Rhodamine 123 as a probe of transmembrane potential in isolated rat-liver mitochondria: spectral and metabolic propertiesPublished by Elsevier ,2003
- Liver cell necrosis: Cellular mechanisms and clinical implicationsGastroenterology, 1995
- Clinical features of an anti-anginal drug in angina pectorisEuropean Heart Journal, 1993
- Antioxidant enzyme status of ischemic and postischemic liver and ischemic kidney in ratsFree Radical Biology & Medicine, 1993
- Contribution of No-reflow phenomenon to hepatic injury after ischemia-reperfusion: Evidence for a role for superoxide anionHepatology, 1992
- Inhibition of Ca2+-induced large-amplitude swelling of liver and heart mitochondria by cyclosporin is probably caused by the inhibitor binding to mitochondrial-matrix peptidyl-prolyl cis-trans isomerase and preventing it interacting with the adenine nucleotide translocaseBiochemical Journal, 1990
- Mitochondria and xanthine oxidase both generate reactive oxygen species in isolated perfused rat liver after hypoxic injuryBiochemical and Biophysical Research Communications, 1989
- Production of free radicals and lipid peroxides in early experimental myocardial ischemiaJournal of Molecular and Cellular Cardiology, 1983
- [15] Isolation of liver or kidney mitochondriaPublished by Elsevier ,1967
- PROTEIN MEASUREMENT WITH THE FOLIN PHENOL REAGENTJournal of Biological Chemistry, 1951