Activation of the c-Jun N-terminal Kinase Signaling Cascade Mediates the Effect of Amyloid-β on Long Term Potentiation and Cell Death in Hippocampus
Open Access
- 1 July 2003
- journal article
- Published by Elsevier
- Vol. 278 (30) , 27971-27980
- https://doi.org/10.1074/jbc.m302530200
Abstract
No abstract availableKeywords
This publication has 56 references indexed in Scilit:
- Alzheimer Amyloid β-Peptide Inhibits the Late Phase of Long-Term Potentiation through Calcineurin-Dependent Mechanisms in the Hippocampal Dentate GyrusNeurobiology of Learning and Memory, 2002
- Naturally secreted oligomers of amyloid β protein potently inhibit hippocampal long-term potentiation in vivoNature, 2002
- Interleukin-1, neuroinflammation, and Alzheimer's diseaseNeurobiology of Aging, 2001
- SB203580, the p38 mitogen-activated protein kinase inhibitor blocks the inhibitory effect of β-amyloid on long-term potentiation in the rat hippocampusNeuroscience Letters, 2000
- E2F1 Mediates Death of B-amyloid-treated Cortical Neurons in a Manner Independent of p53 and Dependent on Bax and Caspase 3Journal of Biological Chemistry, 2000
- Impairment of hippocampal long-term potentiation by Alzheimer amyloid ?-peptidesJournal of Neuroscience Research, 2000
- c‐Jun Contributes to Amyloid β‐Induced Neuronal Apoptosis but Is Not Necessary for Amyloid β‐Induced c‐jun InductionJournal of Neurochemistry, 1999
- Mechanisms of Neuronal Degeneration in Alzheimer's DiseaseNeuron, 1996
- Cell-autonomous Fas (CD95)/Fas-ligand interaction mediates activation-induced apoptosis in T-cell hybridomasNature, 1995
- A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye bindingAnalytical Biochemistry, 1976