Resveratrol Reverses Tumor-Promoter-Induced Inhibition of Gap-Junctional Intercellular Communication
- 1 September 2000
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 275 (3) , 804-809
- https://doi.org/10.1006/bbrc.2000.3378
Abstract
No abstract availableKeywords
This publication has 24 references indexed in Scilit:
- The Cancer Chemopreventive Agent Resveratrol Is Incorporated into Model Membranes and Inhibits Protein Kinase C α ActivityArchives of Biochemistry and Biophysics, 1999
- Resveratrol Preferentially Inhibits Protein Kinase C-Catalyzed Phosphorylation of a Cofactor-Independent, Arginine-Rich Protein Substrate by a Novel MechanismBiochemistry, 1999
- Resveratrol suppresses cell transformation and induces apoptosis through a p53-dependent pathwayCarcinogenesis: Integrative Cancer Research, 1999
- Resveratrol Arrests the Cell Division Cycle at S/G2 Phase TransitionBiochemical and Biophysical Research Communications, 1998
- Resveratrol Inhibits Cyclooxygenase-2 Transcription and Activity in Phorbol Ester-treated Human Mammary Epithelial CellsJournal of Biological Chemistry, 1998
- Recent Advances in Chemoprevention of CancerScience, 1997
- Resveratrol: A molecule whose time has come? And gone?Clinical Biochemistry, 1997
- Cancer Chemopreventive Activity of Resveratrol, a Natural Product Derived from GrapesScience, 1997
- COMMENTARY: Role of connexin genes in growth controlCarcinogenesis: Integrative Cancer Research, 1996
- Changes in gap‐junction permeability, phosphorylation, and number mediated by phorbol ester and non‐phorbol‐ester tumor promoters in rat liver epithelial cellsMolecular Carcinogenesis, 1994