Pink1, Parkin, DJ-1 and mitochondrial dysfunction in Parkinson's disease
- 1 June 2007
- journal article
- review article
- Published by Elsevier in Current Opinion in Neurobiology
- Vol. 17 (3) , 331-337
- https://doi.org/10.1016/j.conb.2007.04.010
Abstract
No abstract availableKeywords
This publication has 53 references indexed in Scilit:
- Antioxidants protect PINK1 -dependent dopaminergic neurons in DrosophilaProceedings of the National Academy of Sciences, 2006
- Parkin affects mitochondrial function and apoptosis in neuronal and myogenic cellsBiochemical and Biophysical Research Communications, 2006
- Mutational analysis of DJ-1 in Drosophila implicates functional inactivation by oxidative damage and agingProceedings of the National Academy of Sciences, 2006
- A regulated interaction with the UIM protein Eps15 implicates parkin in EGF receptor trafficking and PI(3)K–Akt signallingNature Cell Biology, 2006
- Mitochondrial pathology and muscle and dopaminergic neuron degeneration caused by inactivation of Drosophila Pink1 is rescued by ParkinProceedings of the National Academy of Sciences, 2006
- Diverse Effects of Pathogenic Mutations of Parkin That Catalyze Multiple Monoubiquitylation in VitroJournal of Biological Chemistry, 2006
- α-Synuclein and Parkin Contribute to the Assembly of Ubiquitin Lysine 63-linked Multiubiquitin ChainsJournal of Biological Chemistry, 2005
- Parkin Phosphorylation and Modulation of Its E3 Ubiquitin Ligase ActivityJournal of Biological Chemistry, 2005
- Drosophila parkinmutants have decreased mass and cell size and increased sensitivity to oxygen radical stressDevelopment, 2004
- Cysteine-106 of DJ-1 is the most sensitive cysteine residue to hydrogen peroxide-mediated oxidation in vivo in human umbilical vein endothelial cellsBiochemical and Biophysical Research Communications, 2004