Kainic acid-induced seizures produce necrotic, not apoptotic, neurons with internucleosomal DNA cleavage: implications for programmed cell death mechanisms
- 1 June 2000
- journal article
- Published by Elsevier in Neuroscience
- Vol. 98 (1) , 41-53
- https://doi.org/10.1016/s0306-4522(00)00085-3
Abstract
No abstract availableKeywords
This publication has 63 references indexed in Scilit:
- Caspase-3-dependent neuronal death in the hippocampus following kainic acid treatmentMolecular Brain Research, 1999
- Electron Microscopic Evidence against Apoptosis as the Mechanism of Neuronal Death in Global IschemiaJournal of Neuroscience, 1999
- Differential regulation of apoptosis-related genes in resistant and vulnerable subfields of the rat epileptic hippocampusPublished by Elsevier ,1999
- Apoptosis and proliferation of dentate gyrus neurons after single and intermittent limbic seizuresProceedings of the National Academy of Sciences, 1997
- A cautionary note on the use of the TUNEL stain to determine apoptosisNeuroReport, 1995
- Kainic Acid‐Induced Seizures and Brain Damage in the Rat: Different Effects of NMDA‐ and AMPA Receptor AntagonistsBasic & Clinical Pharmacology & Toxicology, 1993
- Internucleosomal DNA Cleavage Should not be the Sole Criterion for Identifying ApoptosisInternational Journal of Radiation Biology, 1992
- Ketamine, Phencyclidine, and MK‐801 Protect Against Kainic Acid‐Induced Seizure‐Related Brain DamageEpilepsia, 1990
- KAINIC ACID SEIZURES AND THE REVERSIBILITY OF CALCIUM LOADING IN VULNERABLE NEURONS IN THE HIPPOCAMPUSNeuropathology and Applied Neurobiology, 1984
- Anoxic-ischaemic cell change in rat brain light microscopic and fine-structural observationsJournal of the Neurological Sciences, 1972