Inactivation of the 25-Hydroxyvitamin D 1α-Hydroxylase and Vitamin D Receptor Demonstrates Independent and Interdependent Effects of Calcium and Vitamin D on Skeletal and Mineral Homeostasis

This publication has 55 references indexed in Scilit:

• Vitamin D₃ supports osteoclastogenesis via functional vitamin D response element of human RANKL gene promoter
  Journal of Cellular Biochemistry, 2003
• Correction of the abnormal mineral ion homeostasis with a high-calcium, high-phosphorus, high-lactose diet rescues the PDDR phenotype of mice deficient for the 25-hydroxyvitamin D-1α-hydroxylase (CYP27B1)
  Bone, 2003
• Aberrant Growth Plate Development in VDR/RXR Double Null Mutant Mice
  Endocrinology, 2001
• Targeted Inactivation of the 25-Hydroxyvitamin D3-1 -Hydroxylase Gene (CYP27B1) Creates an Animal Model of Pseudovitamin D-Deficiency Rickets
  Endocrinology, 2001
• Tissue-Specific Targeting of the Pthrp Gene: The Generation of Mice with Floxed Alleles
  Endocrinology, 2001
• Osteomalacia in Hyp Mice Is Associated with Abnormal Phex Expression and with Altered Bone Matrix Protein Expression and Deposition
  Endocrinology, 2001
• 25-Hydroxyvitamin D 1α-Hydroxylase: Structure of the Mouse Gene, Chromosomal Assignment, and Developmental Expression
  Journal of Bone and Mineral Research, 2001
• Cloning of the mouse 25-hydroxyvitamin D3-1α-hydroxylase (CYP1α) gene
  Biochimica et Biophysica Acta (BBA) - General Subjects, 2000
• Stimulation of Osteoclast Formation by 1,25-Dihydroxyvitamin D Requires Its Binding to Vitamin D Receptor (VDR) in Osteoblastic Cells: Studies Using VDR Knockout Mice
  Endocrinology, 1999
• A simple micromethod for collagen and total protein determination in formalin-fixed paraffin-embedded sections.
  Journal of Histochemistry & Cytochemistry, 1985