Excitotoxic lesions of the ventral anteroventral third ventricle and pressor responses to central sodium, ouabain and angiotensin II
- 21 February 1997
- journal article
- research article
- Published by Elsevier in Brain Research
- Vol. 749 (1) , 157-160
- https://doi.org/10.1016/s0006-8993(96)01381-9
Abstract
No abstract availableKeywords
This publication has 11 references indexed in Scilit:
- INTEGRATIVE ROLE OF THE LAMINA TERMINALIS IN THE REGULATION OF CARDIOVASCULAR AND BODY FLUID HOMEOSTASISClinical and Experimental Pharmacology and Physiology, 1996
- Comparison of c-fos expression in the lamina terminalis of conscious rats after intravenous or intracerebroventricular angiotensinBrain Research Bulletin, 1995
- Regional suppression by lesions in the anterior third ventricle ofc-fos expression induced by either angiotensin II or hypertonic salineNeuroscience, 1995
- The angiotensin receptor subtype AT1A predominates in rat forebrain areas involved in blood pressure, body fluid homeostasis and neuroendocrine controlMolecular Brain Research, 1995
- Murine embryonal carcinoma-derived neurons survive and mature following transplantation into adult rat striatumNeuroscience, 1994
- Brain ouabain-like activity and the sympathoexcitatory and pressor effects of central sodium in rats.Circulation Research, 1992
- Pressor effect of centrally administered sodium chloride: role of the ventral third ventricle region and the area postremaJournal of the Autonomic Nervous System, 1991
- Localization of angiotensin II receptors along the anteroventral third ventricle area of the rat brainBrain Research, 1987
- Centrally-induced vasopressor responses to sodium-potassium adenosine triphosphatase inhibitor, ouabain, may be mediated via angiotensin II in the anteroventral third ventricle in the brain.Japanese Circulation Journal, 1984
- Regional study of cerebral ventricle sensitive sites to angiotensin IIBrain Research, 1976