DECREASED DEXTROSE TOLERANCE IN ACUTE INFECTIOUS DISEASES

1 December 1932

journal article
research article
Published by American Medical Association (AMA) in Archives of internal medicine (1960)

Vol. 50 (6) , 801-818
https://doi.org/10.1001/archinte.1932.00150190003001

Abstract

Transient glycosuria has been noted in various infectious diseases. Hibbard and Morrissey¹ found it in diphtheria. Cammidge² stated that it occurs in diphtheria, scarlet fever, typhoid fever, influenza, appendicitis, measles and infections with suppuration. Buhl³ described its occurrence in Asiatic cholera. Castellani and Willemore⁴ and Harrison⁵ have found that it may be present in malaria. Cammidge⁶ and Higginson⁷ have designated as "sapraemic glycosuria" that accompanying carbuncle or gangrene. This glycosuria is accompanied by a hyperglycemia according to Hollinger.⁸ A lowered dextrose tolerance was demonstrated by Hamman and Hirschman⁹ in lobar pneumonia and acute tonsillitis. Olmsted and Gay¹⁰ found abnormal blood sugar curves in many conditions including those resulting from acute infectious toxins. Tisdall, Drake and Brown¹¹ noted a derangement of the carbohydrate metabolism in infants with acute infectious diarrhea. In the study of various infectious diseases Labbé and Boulin¹² found an increase in the height of the

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