(CTG)n repeats markedly inhibit differentiation of the C2C12 myoblast cell line: implications for congenital myotonic dystrophy
- 24 February 1999
- journal article
- Published by Elsevier in Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
- Vol. 1453 (2) , 221-229
- https://doi.org/10.1016/s0925-4439(98)00104-5
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- Disruption of Splicing Regulated by a CUG-Binding Protein in Myotonic DystrophyScience, 1998
- Expansion of the myotonic dystrophy CTG repeat reduces expression of the flanking DMAHP geneNature Genetics, 1997
- Trinucleotide repeat expansion at the myotonic dystrophy locus reduces expression of DMAHPNature Genetics, 1997
- Normal levels of DM RNA and myotonin protein kinase in skeletal muscle from adult myotonic dystrophy (DM) patientsBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 1996
- Mice lacking the myotonic dystrophy protein kinase develop a late onset progressive myopathyNature Genetics, 1996
- Abnormal myotonic dystrophy protein kinase levels produce only mild myopathy in miceNature Genetics, 1996
- A metalloprotease-disintegrin participating in myoblast fusionNature, 1995
- Myotonic dystrophy: evidence for a possible dominant-negative RNA mutationHuman Molecular Genetics, 1995
- Plasticity of the Differentiated StateScience, 1985
- Immunochemical analysis of myosin heavy chain during avian myogenesis in vivo and in vitro.The Journal of cell biology, 1982