Genetic Manipulation of the Rabbit Heart via Transgenesis
- 11 April 2000
- journal article
- research article
- Published by Wolters Kluwer Health in Circulation
- Vol. 101 (14) , 1715-1721
- https://doi.org/10.1161/01.cir.101.14.1715
Abstract
Background—Transgenesis using cardiac-specific expression has been valuable in exploring cardiac structure-function relationships. To date, cardiac-selective studies have been confined to the mouse...Keywords
This publication has 26 references indexed in Scilit:
- Cardiac troponin T mutations result in allele-specific phenotypes in a mouse model for hypertrophic cardiomyopathyJournal of Clinical Investigation, 1999
- A mouse model of myosin binding protein C human familial hypertrophic cardiomyopathy.Journal of Clinical Investigation, 1998
- A truncated cardiac troponin T molecule in transgenic mice suggests multiple cellular mechanisms for familial hypertrophic cardiomyopathy.Journal of Clinical Investigation, 1998
- Altering Cardiac Function via Transgenesis A Nuts and Bolts ApproachTrends in Cardiovascular Medicine, 1997
- A Mouse Model of Familial Hypertrophic CardiomyopathyScience, 1996
- Anin vivo analysis of transcriptional elements in the mouse α-myosin heavy chain gene promoterTransgenic Research, 1995
- In Vivo Definition of a Cardiac Specific Promoter and Its Potential Utility in Remodeling the HeartAnnals of the New York Academy of Sciences, 1995
- Genes encoding a mouse monoclonal antibody are expressed in transgenic mice, rabbits and pigsGene, 1991
- Production of transgenic rabbits, sheep and pigs by microinjectionNature, 1985
- Regulation of myosin synthesis by thyroid hormone: relative change in the .alpha.- and .beta.-myosin heavy chain mRNA levels in rabbit heartBiochemistry, 1984