DMSO and retinoic acid induce HL-60 differentiation by different but converging pathways
- 1 September 1990
- journal article
- research article
- Published by Elsevier in Experimental Cell Research
- Vol. 190 (1) , 137-140
- https://doi.org/10.1016/0014-4827(90)90155-4
Abstract
No abstract availableKeywords
This publication has 12 references indexed in Scilit:
- HL-R5 and HL-D4: Two differentiation resistant HL-60 variantsLeukemia Research, 1988
- Cloning and expression of two human genes encoding calcium-binding proteins that are regulated during myeloid differentiation.Molecular and Cellular Biology, 1988
- An oligomer complementary to c-myc mRNA inhibits proliferation of HL-60 promyelocytic cells and induces differentiation.Molecular and Cellular Biology, 1988
- The HL-60 promyelocytic leukemia cell line: proliferation, differentiation, and cellular oncogene expressionBlood, 1987
- A block to elongation is largely responsible for decreased transcription of c-myc in differentiated HL60 cellsNature, 1986
- Transcriptional arrest within the first exon is a fast control mechanism in c-myc gene expressionNucleic Acids Research, 1986
- Stimulation of 3T3 cells induces transcription of the c-fos proto-oncogeneNature, 1984
- Regulation of myc gene expression in HL-60 leukaemia cells by a vitamin D metaboliteNature, 1983
- A technique for radiolabeling DNA restriction endonuclease fragments to high specific activityAnalytical Biochemistry, 1983
- onc gene amplification in promyelocytic leukaemia cell line HL-60 and primary leukaemic cells of the same patientNature, 1982