DNA damage checkpoint and repair centers
- 17 April 2004
- journal article
- review article
- Published by Elsevier in Current Opinion in Cell Biology
- Vol. 16 (3) , 328-334
- https://doi.org/10.1016/j.ceb.2004.03.011
Abstract
No abstract availableKeywords
This publication has 64 references indexed in Scilit:
- Senescing human cells and ageing mice accumulate DNA lesions with unrepairable double-strand breaksNature Cell Biology, 2004
- Repair of DNA double strand breaks by non-homologous end joiningBiochimie, 2003
- A DNA damage checkpoint response in telomere-initiated senescenceNature, 2003
- Colocalization of multiple DNA double-strand breaks at a single Rad52 repair centreNature Cell Biology, 2003
- Molecular Anatomy of the DNA Damage and Replication CheckpointsRadiation Research, 2003
- Role ofRAD52Epistasis Group Genes in Homologous Recombination and Double-Strand Break RepairMicrobiology and Molecular Biology Reviews, 2002
- Histone H2AX Is Phosphorylated in an ATR-dependent Manner in Response to Replicational StressJournal of Biological Chemistry, 2001
- Nuclear foci of mammalian recombination proteins are located at single-stranded DNA regions formed after DNA damageProceedings of the National Academy of Sciences, 1999
- Nuclear foci of mammalian Rad51 recombination protein in somatic cells after DNA damage and its localization in synaptonemal complexes.Proceedings of the National Academy of Sciences, 1995
- RecA homologs Dmc1 and Rad51 interact to form multiple nuclear complexes prior to meiotic chromosome synapsisCell, 1994