Structural alterations in the peptide backbone of beta-amyloid core protein may account for its deposition and stability in Alzheimer's disease.
Open Access
- 1 February 1993
- journal article
- abstracts
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 268 (5) , 3072-3083
- https://doi.org/10.1016/s0021-9258(18)53661-9
Abstract
No abstract availableKeywords
This publication has 72 references indexed in Scilit:
- On the Other Hand . . .Science, 1992
- Covalent modification of Alzheimer's amyloid β-peptide in formic acid solutionsBiochemical and Biophysical Research Communications, 1992
- Exact cleavage site of Alzheimer amyloid precursor in neuronal PC-12 cellsNeuroscience Letters, 1991
- Processing of Alzheimer's disease-associated β-amyloid precursor proteinJournal of Molecular Neuroscience, 1990
- Enzymatic methyl esterification of a deamidated form of mouse epidermal growth factorInternational Journal of Peptide and Protein Research, 1989
- Complement activation in amyloid plaques in Alzheimer’s dementiaVirchows Archiv B Cell Pathology Including Molecular Pathology, 1988
- Two major isozymes of the protein D-aspartyl/L-isoaspartyl methyltransferase from human erythrocytesBiochemical and Biophysical Research Communications, 1988
- Deamidation of the asparaginyl‐glycyl sequenceInternational Journal of Peptide and Protein Research, 1986
- Alzheimer's disease: Initial report of the purification and characterization of a novel cerebrovascular amyloid proteinBiochemical and Biophysical Research Communications, 1984
- New approach to the mass spectroscopy of non-volatile compoundsBiochemical and Biophysical Research Communications, 1974