Molecular mechanism of Mitomycin C-dependent caspase-8 regulation: implications for apoptosis and synergism with interferon-α signalling
- 6 October 2007
- journal article
- research article
- Published by Springer Nature in Apoptosis
- Vol. 12 (12) , 2259-2270
- https://doi.org/10.1007/s10495-007-0145-x
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- Death effector domain DEDa, a self-cleaved product of caspase-8/Mch5, translocates to the nucleus by binding to ERK1/2 and upregulates procaspase-8 expression via a p53-dependent mechanismThe EMBO Journal, 2007
- The role of JNK2 in toxic liver injuryJournal of Hepatology, 2006
- Tumor Necrosis Factor-induced Toxic Liver Injury Results from JNK2-dependent Activation of Caspase-8 and the Mitochondrial Death PathwayJournal of Biological Chemistry, 2006
- Interferon-α enhances TRAIL-mediated apoptosis by up-regulating caspase-8 transcription in human hepatoma cellsJournal of Hepatology, 2006
- Mitomycin-C Induces the Apoptosis of Human Tenon’s Capsule Fibroblast by Activation ofc-JunN-Terminal Kinase 1 and Caspase-3 ProteaseInvestigative Opthalmology & Visual Science, 2005
- The Human Caspase-8 Promoter Sustains Basal Activity through SP1 and ETS-like Transcription Factors and Can Be Up-regulated by a p53-dependent MechanismJournal of Biological Chemistry, 2003
- Jun kinase modulates tumor necrosis factor-dependent apoptosis in liver cellsHepatology, 2002
- Futile Caspase-8 Activation during the Apoptotic Cell Death Induced by DNA Damaging Agents in Human B-LymphoblastsExperimental Cell Research, 2001
- FLICE, A Novel FADD-Homologous ICE/CED-3–like Protease, Is Recruited to the CD95 (Fas/APO-1) Death-Inducing Signaling ComplexCell, 1996
- Involvement of MACH, a Novel MORT1/FADD-Interacting Protease, in Fas/APO-1- and TNF Receptor–Induced Cell DeathCell, 1996