REDUCED EXPRESSION OF FLICE-INHIBITORY PROTEIN (FLIP) AND NFκB IS ASSOCIATED WITH DEATH RECEPTOR-INDUCED CELL DEATH IN HUMAN AORTIC ENDOTHELIAL CELLS (HAECs)
- 31 July 2001
- Vol. 15 (2) , 66-74
- https://doi.org/10.1006/cyto.2001.0916
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Hepatocytes Sensitized to Tumor Necrosis Factor-α Cytotoxicity Undergo Apoptosis through Caspase-dependent and Caspase-independent PathwaysPublished by Elsevier ,2000
- NF-κB Activation Is Required for Human Endothelial Survival during Exposure to Tumor Necrosis Factor-α but Not to Interleukin-1β or LipopolysaccharidePublished by Elsevier ,1999
- Viral FLICE-inhibitory proteins (FLIPs) prevent apoptosis induced by death receptorsNature, 1997
- Dissection of TNF Receptor 1 Effector Functions: JNK Activation Is Not Linked to Apoptosis While NF-κB Activation Prevents Cell DeathCell, 1996
- FLICE, A Novel FADD-Homologous ICE/CED-3–like Protease, Is Recruited to the CD95 (Fas/APO-1) Death-Inducing Signaling ComplexCell, 1996
- Involvement of MACH, a Novel MORT1/FADD-Interacting Protease, in Fas/APO-1- and TNF Receptor–Induced Cell DeathCell, 1996
- TNF-Dependent Recruitment of the Protein Kinase RIP to the TNF Receptor-1 Signaling ComplexImmunity, 1996
- Pharmacological inhibition of programmed lymphocyte deathImmunology Today, 1994
- Two TNF receptorsImmunology Today, 1992
- Characterization of binding and biological effects of monoclonal antibodies against a human tumor necrosis factor receptor.The Journal of Experimental Medicine, 1990