Intrahippocampal or intraamygdala infusion of KN62, a specific inhibitor of calcium/calmodulin-dependent protein kinase II, causes retrograde amnesia in the rat
- 31 May 1994
- journal article
- research article
- Published by Elsevier in Behavioral and Neural Biology
- Vol. 61 (3) , 203-205
- https://doi.org/10.1016/s0163-1047(05)80001-9
Abstract
No abstract availableKeywords
This publication has 9 references indexed in Scilit:
- Post-training intrahippocampal infusion of protein kinase C inhibitors causes amnesia in ratsBehavioral and Neural Biology, 1994
- Long‐term potentiation and the mechanisms of memoryDrug Development Research, 1993
- A synaptic model of memory: long-term potentiation in the hippocampusNature, 1993
- Postsynaptic then presynaptic protein kinase C activity may be necessary for long-term potentiationNeuroscience, 1992
- Deficient Hippocampal Long-Term Potentiation in α-Calcium-Calmodulin Kinase II Mutant MiceScience, 1992
- Impaired Spatial Learning in α-Calcium-Calmodulin Kinase II Mutant MiceScience, 1992
- Effects of KN-62, a specific inhibitor of calcium/calmodulin-dependent protein kinase II, on long-term potentiation in the rat hippocampusNeuroscience Letters, 1991
- KN-62, 1-[N,O-bis(5-isoquinolinesulfonyl)-N-methyl-L-tyrosyl]-4-phenylpiperazi ne, a specific inhibitor of Ca2+/calmodulin-dependent protein kinase II.Journal of Biological Chemistry, 1990
- Polymyxin B, an inhibitor of protein kinase C, prevents the maintenance of synaptic long-term potentiation in hippocampal CA1 neuronsBrain Research, 1988