gp120 as an etiologic agent for NeuroAIDS: Neurotoxicity and model systems
- 31 December 1994
- journal article
- review article
- Published by Elsevier in Advances in Neuroimmunology
- Vol. 4 (3) , 157-165
- https://doi.org/10.1016/s0960-5428(06)80252-4
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- Cytokine expression in the brain during the acquired immunodeficiency syndromeAnnals of Neurology, 1992
- Neocortical damage during HIV infectionAnnals of Neurology, 1991
- Vasoactive intestinal peptide: A neurotrophic releasing agent and an astroglial mitogenJournal of Neuroscience Research, 1990
- Neuronal cell killing by the envelope protein of HIV and its prevention by vasoactive intestinal peptideNature, 1988
- Tritiated D-ala1-peptide T binding: A pharmacologic basis for the design of drugs which inhibit HIV receptor bindingDrug Development Research, 1988
- Peptide t sequences prevent neuronal cell death produced by the envelope protein (gp 120) of the human immunodeficiency virusDrug Development Research, 1988
- CD4 receptor binding peptides that block HIV infectivity cause human monocyte chemotaxisFEBS Letters, 1987
- Vasoactive intestinal peptide 1‐12: A ligand for the CD4 (T4)/human immunodeficiency virus receptorJournal of Neuroscience Research, 1987
- Shedding and Interspecies Type Sero-reactivity of the Envelope Glycopolypeptide gp120 of the Human Immunodeficiency VirusJournal of General Virology, 1986
- T-lymphocyte T4 molecule behaves as the receptor for human retrovirus LAVNature, 1984