Alteration of HERG Current Profile during the Cardiac Ventricular Action Potential, Following a Pore Mutation
- 30 December 1998
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 253 (3) , 719-724
- https://doi.org/10.1006/bbrc.1998.9837
Abstract
No abstract availableKeywords
This publication has 10 references indexed in Scilit:
- Time course and voltage dependence of expressed HERG current compared with native ”rapid" delayed rectifier K current during the cardiac ventricular action potentialPflügers Archiv - European Journal of Physiology, 1998
- A mutation in the pore region of HERG K+ channels expressed in Xenopus oocytes reduces rectification by shifting the voltage dependence of inactivationThe Journal of Physiology, 1998
- Properties of HERG Channels Stably Expressed in HEK 293 Cells Studied at Physiological TemperatureBiophysical Journal, 1998
- Molecular determinants for activation and inactivation of HERG, a human inward rectifier potassium channel.The Journal of Physiology, 1996
- The inward rectification mechanism of the HERG cardiac potassium channelNature, 1996
- HERG, a Human Inward Rectifier in the Voltage-Gated Potassium Channel FamilyScience, 1995
- Two Components of the Delayed Rectifier K + Current in Ventricular Myocytes of the Guinea Pig TypeCirculation Research, 1995
- A mechanistic link between an inherited and an acquird cardiac arrthytmia: HERG encodes the IKr potassium channelCell, 1995
- A molecular basis for cardiac arrhythmia: HERG mutations cause long QT syndromeCell, 1995
- Two components of cardiac delayed rectifier K+ current. Differential sensitivity to block by class III antiarrhythmic agents.The Journal of general physiology, 1990