Nephrotoxin exposure in utero reduces glomerular number in sclerosis-prone but not sclerosis-resistant mice

This publication has 12 references indexed in Scilit:

• Nature and Severity of the Glomerular Response to Nephron Reduction Is Strain-Dependent in Mice
  The American Journal of Pathology, 1999
• Strain differences rather than hyperglycemia determine the severity of glomerulosclerosis in mice
  Kidney International, 1998
• Early defect in branching morphogenesis of the ureteric bud in induced nephron deficit
  Kidney International, 1996
• Dissociation of glomerular hypertrophy, cell proliferation, and glomerulosclerosis in mouse strains heterozygous for a mutation (Os) which induces a 50% reduction in nephron number.
  Journal of Clinical Investigation, 1996
• Intrauterine growth retardation leads to a permanent nephron deficit in the rat
  Pediatric Nephrology, 1994
• The effect of intrauterine growth retardation on the development of renal nephrons
  BJOG: An International Journal of Obstetrics and Gynaecology, 1992
• Long-Term Effects of Mild Oligonephronia Induced In Utero by Gentamicin in the Rat
  Pediatric Research, 1991
• Effects of prenatal and postnatal exposure to gentamicin on renal differentiation in the rat
  Toxicology, 1987
• Transferrin as a fetal growth factor: acquisition of responsiveness related to embryonic induction.
  Proceedings of the National Academy of Sciences, 1983
• Congenital Bilateral Oligonephronic Renal Hypoplasia with Hypertrophy of Nephrons (Oligoméganéphronie): Studies by Microdissection
  American Journal of Clinical Pathology, 1969