STIMULATION OF PROSTACYCLIN BIOSYNTHETIC ACTIVITY BY ESTRADIOL IN RAT AORTIC SMOOTH-MUSCLE CELLS IN CULTURE

  • 1 January 1980
    • journal article
    • research article
    • Vol. 619  (1) , 107-118
Abstract
Effects of estradiol on cell proliferation and prostacyclin biosynthetic activity in cultured rat aortic smooth muscle cells are investigated. Prostacyclin is the major product formed from arachidonic acid by the cells, in a cell-free homogenate system and in intact cells. When cells are treated with estradiol, no effect on cell proliferation is observed but a significantly stimulatory effect on prostacyclin biosynthetic activity in cells is evident. The maximal effect of estradiol on the stimulation of prostacyclin biosynthetic activity is observed after 5 day treatment of the cells with estradiol at its physiological concentration (10-9 M). Estradiol would be a protective hormone in atherosclerotic heart disease.