Amyloid Phenotype Characterization of Transgenic Mice Overexpressing both Mutant Amyloid Precursor Protein and Mutant Presenilin 1 Transgenes

This publication has 20 references indexed in Scilit:

• Accelerated Alzheimer-type phenotype in transgenic mice carrying both mutant amyloid precursor protein and presenilin 1 transgenes
  Nature Medicine, 1998
• Accelerated Amyloid Deposition in the Brains of Transgenic Mice Coexpressing Mutant Presenilin 1 and Amyloid Precursor Proteins
  Neuron, 1997
• Mutant presenilins of Alzheimer's disease increase production of 42-residue amyloid β-protein in both transfected cells and transgenic mice
  Nature Medicine, 1997
• Familial Alzheimer's Disease–Linked Presenilin 1 Variants Elevate Aβ1–42/1–40 Ratio In Vitro and In Vivo
  Neuron, 1996
• Increased amyloid-β42(43) in brains of mice expressing mutant presenilin 1
  Nature, 1996
• Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein
  Nature, 1995
• Release of Excess Amyloid β Protein from a Mutant Amyloid β Protein Precursor
  Science, 1993
• Mutation of the β-amyloid precursor protein in familial Alzheimer's disease increases β-protein production
  Nature, 1992
• Transection of the rat olfactory nerve increases glial fibrillary acidic protein immunoreactivity from the olfactory bulb to the piriform cortex
  Glia, 1990
• Short- and long-term consequences of intracranial injections of the excitotoxin, quinolinic acid, as evidenced by GFA immunohistochemistry of astrocytes
  Brain Research, 1986