β-Amyloid-(1–42) Impairs Activity-dependent cAMP-response Element-binding Protein Signaling in Neurons at Concentrations in Which Cell Survival Is Not Compromised
Open Access
- 1 May 2001
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 276 (20) , 17301-17306
- https://doi.org/10.1074/jbc.m010450200
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- Neuronal Apoptosis Induced by β-Amyloid Is Mediated by Caspase-8Neurobiology of Disease, 1999
- Soluble Amyloid β Peptide Concentration as a Predictor of Synaptic Change in Alzheimer's DiseaseThe American Journal of Pathology, 1999
- Multiple Pathways of Apoptosis in PC12 Cells: CrmA INHIBITS APOPTOSIS INDUCED BY β-AMYLOIDPublished by Elsevier ,1999
- TRANSCRIPTIONAL REGULATION BY CYCLIC AMPAnnual Review of Biochemistry, 1997
- Elevated Amyloid β Protein(1-40) Level Induces CREB Phosphorylation at Serine-133 via p44/42 MAP Kinase (Erk1/2)-Dependent Pathway in Rat Pheochromocytoma PC12 CellsBiochemical and Biophysical Research Communications, 1997
- The Release of Cytochrome c from Mitochondria: A Primary Site for Bcl-2 Regulation of ApoptosisScience, 1997
- Mechanisms of Neuronal Degeneration in Alzheimer's DiseaseNeuron, 1996
- Deficient long-term memory in mice with a targeted mutation of the cAMP-responsive element-binding proteinCell, 1994
- Induction of a dominant negative CREB transgene specifically blocks long-term memory in DrosophilaCell, 1994
- Release of Excess Amyloid β Protein from a Mutant Amyloid β Protein PrecursorScience, 1993