Mechanisms of Asbestos-induced Nitric Oxide Production by Rat Alveolar Macrophages in Inhalation and in vitro Models 11Supported by grants from NHLBI (RO1 HL39469) and NIEHS (RO1 03878).
- 1 March 1998
- journal article
- Published by Elsevier in Free Radical Biology & Medicine
- Vol. 24 (5) , 778-788
- https://doi.org/10.1016/s0891-5849(97)00357-2
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- Differential induction of c-fos, c-jun, and apoptosis in lung epithelial cells exposed to ROS or RNSAmerican Journal of Physiology-Lung Cellular and Molecular Physiology, 1997
- Nitric oxide increases cellular glutathione levels in rat lung fibroblasts.American Journal of Respiratory Cell and Molecular Biology, 1995
- Induction of c-fos and c-jun proto-oncogenes in target cells of the lung and pleura by carcinogenic fibers.American Journal of Respiratory Cell and Molecular Biology, 1994
- Dose-responsive increases in pulmonary fibrosis after inhalation of asbestos.American Journal of Respiratory and Critical Care Medicine, 1994
- Modulation of acute inflammation by endogenous nitric oxideEuropean Journal of Pharmacology, 1992
- Nitric oxide as an antioxidantArchives of Biochemistry and Biophysics, 1991
- Role of asbestos and other fibres in the development of diffuse malignant mesothelioma.Thorax, 1990
- Apparent hydroxyl radical production by peroxynitrite: implications for endothelial injury from nitric oxide and superoxide.Proceedings of the National Academy of Sciences, 1990
- Asbestos: Scientific Developments and Implications for Public PolicyScience, 1990
- Endothelium-dependent relaxation in rat aorta may be mediated through cyclic GMP-dependent protein phosphorylationNature, 1983