Moderate Posttraumatic Hypothermia Decreases Early Calpain-Mediated Proteolysis and Concomitant Cytoskeletal Compromise in Traumatic Axonal Injury
- 1 September 1999
- journal article
- Published by Elsevier in Experimental Neurology
- Vol. 159 (1) , 319-328
- https://doi.org/10.1006/exnr.1999.7139
Abstract
No abstract availableKeywords
This publication has 70 references indexed in Scilit:
- Posttraumatic hypothermia in the treatment of axonal damage in an animal model of traumatic axonal injuryJournal of Neurosurgery, 1998
- Simultaneous Degradation of αII- and βII-Spectrin by Caspase 3 (CPP32) in Apoptotic CellsJournal of Biological Chemistry, 1998
- Subcellular Localization and Duration of μ-Calpain and m-Calpain Activity after Traumatic Brain Injury in the Rat: A Casein Zymography StudyJournal of Cerebral Blood Flow & Metabolism, 1998
- Immunohistochemical Study of Calpain-Mediated Breakdown Products to α-Spectrin Following Controlled Cortical Impact Injury in the RatJournal of Neurotrauma, 1997
- Hyperthermia enhances spectrin breakdown in transient focal cerebral ischemiaBrain Research, 1997
- Treatment of Experimental Brain Injury with Moderate Hypothermia and 21-AminosteroidsJournal of Neurotrauma, 1996
- Time-Related Neuronal Changes following Middle Cerebral Artery Occlusion: Implications for Therapeutic Intervention and the Role of CalpainJournal of Cerebral Blood Flow & Metabolism, 1995
- Role of calpain in spinal cord injury: increased mcalpain immunoreactivity in spinal cord after compression injury in the ratNeurochemistry International, 1995
- Effects of normothermic versus mild hyperthermic forebrain ischemia in rats.Stroke, 1990
- Postischemic moderate hypothermia inhibits CA1 hippocampal ischemic neuronal injuryNeuroscience Letters, 1989