Acetaminophen-induced cytotoxicity in cultured mouse hepatocytes: Effects of Ca2+-endonuclease, DNA repair, and glutathione depletion inhibitors on DNA fragmentation and cell death

This publication has 43 references indexed in Scilit:

• The role of calcium in lethal cell injury
  Chemical Research in Toxicology, 1990
• Early loss of large genomic DNA in vivo with accumulation of Ca2+ in the nucleus during acetaminophen-induced liver injury
  Toxicology and Applied Pharmacology, 1990
• Immediate rise in intracellular calcium and glycogen phosphorylase a activities upon acetaminophen covalent binding leading to hepatotoxicity in mice
  Toxicology, 1988
• Structural requirements for cytoprotective agents in galactosamine-induced hepatic necrosis*1
  Toxicology and Applied Pharmacology, 1985
• Comparative influences of different PB-type and 3-MC-type polychlorinated biphenyl-induced phenotypes on cytocidal hepatotoxicity of bromobenzene and acetaminophen
  Toxicology and Applied Pharmacology, 1984
• Apparent maintenance of cytochrome P 450 by nicotinamide in primary cultures of rat hepatocytes
  Life Sciences, 1979
• Inhibition of rat liver Ca2+, Mg2+-dependent endonuclease activity by nicotinamide adenine dinucleotide and poly(adenosine diphosphate ribose) synthetase
  Biochemical and Biophysical Research Communications, 1974
• The calcium dependent endonuclease activity of isolated nuclear preparations. Relationships between its occurrence and the occurrence of other classes of enzymes found in nuclear preparations
  Biochemical and Biophysical Research Communications, 1973
• Inhibition of transfer factor Ts by aurintricarboxylic acid
  Biochemical and Biophysical Research Communications, 1970
• Isolation of pure and unaltered liver nuclei morphology and biochemical composition
  Experimental Cell Research, 1956