Apoptosis induced by endoplasmic reticulum stress depends on activation of caspase-3 via caspase-12
- 3 February 2004
- journal article
- research article
- Published by Elsevier in Neuroscience Letters
- Vol. 357 (2) , 127-130
- https://doi.org/10.1016/j.neulet.2003.12.080
Abstract
No abstract availableKeywords
This publication has 15 references indexed in Scilit:
- BAX and BAK Regulation of Endoplasmic Reticulum Ca 2+ : A Control Point for ApoptosisScience, 2003
- Transmission of cell stress from endoplasmic reticulum to mitochondriaThe Journal of cell biology, 2002
- Coupling Endoplasmic Reticulum Stress to the Cell Death ProgramJournal of Biological Chemistry, 2002
- Human caspase 12 has acquired deleterious mutationsBiochemical and Biophysical Research Communications, 2002
- Coupling Endoplasmic Reticulum Stress to the Cell Death ProgramJournal of Biological Chemistry, 2001
- Increased Production of β-Amyloid and Vulnerability to Endoplasmic Reticulum Stress by an Aberrant Spliced Form of Presenilin 2Journal of Biological Chemistry, 2001
- Caspase-12 mediates endoplasmic-reticulum-specific apoptosis and cytotoxicity by amyloid-βNature, 2000
- Caspases: Enemies WithinScience, 1998
- Induction of Apoptotic Program in Cell-Free Extracts: Requirement for dATP and Cytochrome cPublished by Elsevier ,1996
- The presence of malfolded proteins in the endoplasmic reticulum signals the induction of glucose-regulated proteinsNature, 1988