Lovastatin ameliorates α-synuclein accumulation and oxidation in transgenic mouse models of α-synucleinopathies
- 26 November 2009
- journal article
- Published by Elsevier in Experimental Neurology
- Vol. 221 (2) , 267-274
- https://doi.org/10.1016/j.expneurol.2009.11.015
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- Biophysics of Parkinsons Disease: Structure and Aggregation of α- SynucleinCurrent Protein & Peptide Science, 2009
- Lipid Rafts Mediate the Synaptic Localization of α-SynucleinJournal of Neuroscience, 2004
- Oligomers on the Brain: the Emerging Role of Soluble Protein Aggregates in Neurodegeneration.Protein & Peptide Letters, 2004
- Oxidative Modifications of α‐SynucleinAnnals of the New York Academy of Sciences, 2003
- Pathogenesis of parkinson's disease: dopamine, vesicles and α-synucleinNature Reviews Neuroscience, 2002
- Statins, 3-Hydroxy-3-Methylglutaryl Coenzyme A Reductase Inhibitors, Are Able to Reduce Superoxide Anion Production by NADPH Oxidase in THP-1-Derived MonocytesJournal of Cardiovascular Pharmacology, 2002
- Differential neuropathological alterations in transgenic mice expressing α‐synuclein from the platelet‐derived growth factor and Thy‐1 promotersJournal of Neuroscience Research, 2002
- Induction of α-Synuclein Aggregation by Intracellular Nitrative InsultJournal of Neuroscience, 2001
- β-Amyloid peptides enhance α-synuclein accumulation and neuronal deficits in a transgenic mouse model linking Alzheimer's disease and Parkinson's diseaseProceedings of the National Academy of Sciences, 2001
- Dopaminergic Loss and Inclusion Body Formation in α-Synuclein Mice: Implications for Neurodegenerative DisordersScience, 2000