Age increases axon loss associated with primary demyelination in cuprizone-induced demyelination in C57BL/6 mice
- 30 June 2006
- journal article
- research article
- Published by Elsevier in Journal of Neuroimmunology
- Vol. 175 (1-2) , 69-76
- https://doi.org/10.1016/j.jneuroim.2006.03.002
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Patients lacking the major CNS myelin protein, proteolipid protein 1, develop length-dependent axonal degeneration in the absence of demyelination and inflammationBrain, 2002
- Oedema and glial cell involvement in the aged mouse brain after permanent focal ischaemiaNeuropathology and Applied Neurobiology, 2000
- Acute axonal injury in multiple sclerosis: Correlation with demyelination and inflammationBrain, 2000
- Axonal damage in acute multiple sclerosis lesionsBrain, 1997
- Two models of multiple sclerosis: Experimental allergic encephalomyelitis (EAE) and theiler's murine encephalomyelitis virus (TMEV) infection. A pathological and immunological comparisonMicroscopy Research and Technique, 1995
- Reactive mononuclear phagocytes release neurotoxins after ischemic and traumatic injury to the central nervous systemJournal of Neuroscience Research, 1993
- Remyelination of the superior cerebellar peduncle in old mice following demyelination induced by cuprizoneJournal of the Neurological Sciences, 1974
- Remyelination of the superior cerebellar peduncle in the mouse following demyelination induced by feeding cuprizoneJournal of the Neurological Sciences, 1973
- Demyelination of the superior cerebellar peduncle in the mouse induced by cuprizoneJournal of the Neurological Sciences, 1973
- Observations on oligodendrocyte degeneration, the resolution of status spongiosus and remyelination in cuprizone intoxication in miceJournal of Neurocytology, 1972