Central Pore Residues Mediate the p97/VCP Activity Required for ERAD
- 1 May 2006
- journal article
- Published by Elsevier in Molecular Cell
- Vol. 22 (4) , 451-462
- https://doi.org/10.1016/j.molcel.2006.03.036
Abstract
No abstract availableKeywords
This publication has 52 references indexed in Scilit:
- Nucleotide Dependent Motion and Mechanism of Action of p97/VCPJournal of Molecular Biology, 2005
- Dissection of the Dislocation Pathway for Type I Membrane Proteins with a New Small Molecule Inhibitor, EeyarestatinMolecular Biology of the Cell, 2004
- Distinct Roles for the AAA ATPases NSF and p97 in the Secretory PathwayMolecular Biology of the Cell, 2004
- Chemical cross‐linking and mass spectrometry for mapping three‐dimensional structures of proteins and protein complexesJournal of Mass Spectrometry, 2003
- The crystal structure of murine p97/VCP at 3.6ÅJournal of Structural Biology, 2003
- ATP-binding sites in brain p97/VCP (valosin-containing protein), a multifunctional AAA ATPaseBiochemical Journal, 2003
- Functional Analysis of the Trypanosomal AAA ProteinTbVCP with trans-Dominant ATP Hydrolysis MutantsPublished by Elsevier ,2001
- Endoplasmic Reticulum (ER)-associated Degradation of T Cell Receptor SubunitsJournal of Biological Chemistry, 2001
- The Role of Multiubiquitination in Dislocation and Degradation of the α Subunit of the T Cell Antigen ReceptorJournal of Biological Chemistry, 1999
- The α Chain of the T Cell Antigen Receptor Is Degraded in the CytosolImmunity, 1997