Extracellular phospholipase A2: causative agent in circulatory collapse of septic shock?
- 1 July 1988
- journal article
- Published by Springer Nature in Inflammation Research
- Vol. 24 (3-4) , 320-325
- https://doi.org/10.1007/bf02028289
Abstract
No abstract availableKeywords
This publication has 41 references indexed in Scilit:
- Pathogenesis of hypotension in septic shockCritical Care Medicine, 1988
- Potential therapeutic efficacy of inhibitors of human phospholipase A2 in septic shockInflammation Research, 1986
- Shock and Tissue Injury Induced by Recombinant Human CachectinScience, 1986
- Inflammatory Effect of Intradermal Administration of Soluble Phospholipase A2 in RabbitsJournal of Investigative Dermatology, 1986
- Characterization of extracellular phospholipase A2 in rheumatoid synovial fluidLife Sciences, 1985
- The Effects of High-Dose Corticosteroids in Patients with Septic ShockNew England Journal of Medicine, 1984
- Involvement of circulating phospholipase A2 in the pathogenesis of the hemodynamic changes in endotoxin shockCanadian Journal of Physiology and Pharmacology, 1983
- The efficacy of anti-inflammatory agents with respect to extracellular phospholipase A2 activityLife Sciences, 1982
- Phospholipase A2 stimulated release of prostanoids from the isolated, perfused rabbit liver: implications in regional cellular injuryCanadian Journal of Physiology and Pharmacology, 1981
- Extracellular phospholipase A2 mediates inflammatory hyperaemiaNature, 1981