Human Translocation Liposarcoma-CCAAT/Enhancer Binding Protein (C/EBP) Homologous Protein (TLS-CHOP) Oncoprotein Prevents Adipocyte Differentiation by Directly Interfering with C/EBPβ Function
Open Access
- 1 June 1998
- journal article
- Published by Elsevier
- Vol. 273 (25) , 15574-15581
- https://doi.org/10.1074/jbc.273.25.15574
Abstract
No abstract availableKeywords
This publication has 25 references indexed in Scilit:
- 15-Deoxy-Δ12,14-Prostaglandin J2 is a ligand for the adipocyte determination factor PPARγCell, 1995
- Cascade regulation of terminal adipocyte differentiation by three members of the C/EBP family of leucine zipper proteins.Genes & Development, 1995
- Stimulation of adipogenesis in fibroblasts by PPARγ2, a lipid-activated transcription factorCell, 1994
- A novel effector domain from the RNA-binding protein TLS or EWS is required for oncogenic transformation by CHOP.Genes & Development, 1994
- Ectopic expression of the CCAAT/enhancer-binding protein alpha promotes the adipogenic program in a variety of mouse fibroblastic cells.Genes & Development, 1994
- Fusion of the dominant negative transcription regulator CHOP with a novel gene FUS by translocation t(12;16) in malignant liposarcomaNature Genetics, 1993
- Fusion of CHOP to a novel RNA-binding protein in human myxoid liposarcomaNature, 1993
- Reciprocal Regulation of Adipogenesis by Myc and C/EBPαScience, 1992
- A liver-enriched transcriptional activator protein, LAP, and a transcriptional inhibitory protein, LIP, are translated from the sam mRNACell, 1991
- Regulated expression of three C/EBP isoforms during adipose conversion of 3T3-L1 cells.Genes & Development, 1991