Intra-cerebellar infusion of NMDA receptor antagonist AP5 disrupts classical eyeblink conditioning in rabbits
- 19 December 2000
- journal article
- Published by Elsevier in Brain Research
- Vol. 887 (1) , 144-156
- https://doi.org/10.1016/s0006-8993(00)03005-5
Abstract
No abstract availableKeywords
This publication has 39 references indexed in Scilit:
- Cerebellar circuits and synaptic mechanisms involved in classical eyeblink conditioningTrends in Neurosciences, 1997
- Roles of Cerebellar Cortex and Nuclei in Motor Learning: Contradictions or Clues?Neuron, 1997
- Detailed behavioral analysis of water maze acquisition under APV or CNQX: Contribution of sensorimotor disturbances to drug-induced acquisition deficits.Behavioral Neuroscience, 1996
- Early acquisition, but not retention, of the classically conditioned eyeblink response is N-methyl-D-aspartate (NMDA) receptor dependent.Behavioral Neuroscience, 1996
- Intrahippocampal administration of both the d- and the l-isomers of AP5 disrupt spontaneous alternation behavior and evoked potentialsBehavioral and Neural Biology, 1994
- Intra-amygdala infusion of the N-methyl-d-aspartate receptor antagonist AP5 impairs acquisition but not performance of discriminated approach to an appetitive CSBehavioral and Neural Biology, 1994
- Amnesia by post-training infusion of glutamate receptor antagonists into the amygdala, hippocampus, and entorhinal cortexBehavioral and Neural Biology, 1992
- Effects of lidocaine injection in the interpositus nucleus and red nucleus on conditioned behavioral and neuronal responsesBrain Research, 1990
- Blocking of acquisition but not expression of conditioned fear-potentiated startle by NMDA antagonists in the amygdalaNature, 1990
- Synaptic plasticity and learning II: Do different kinds of plasticity underlie different kinds of learning?Neuropsychologia, 1989