Multiple loss of heterozygosity without K-ras mutation identified by molecular analysis on fine-needle aspiration cytology specimen of acinar cell carcinoma of pancreas
- 26 June 2002
- journal article
- case report
- Published by Wiley in Diagnostic Cytopathology
- Vol. 27 (1) , 42-46
- https://doi.org/10.1002/dc.10147
Abstract
No abstract availableKeywords
This publication has 15 references indexed in Scilit:
- Distinguishing de Novo Second Cancer Formation from Tumor Recurrence: Mutational Fingerprinting by Microdissection GenotypingThe Journal of Molecular Diagnostics, 2001
- Pancreatic tumours: molecular pathways implicated in ductal cancer are involved in ampullary but not in exocrine nonductal or endocrine tumorigenesisBritish Journal of Cancer, 2001
- Allelotype of pancreatic acinar cell carcinomaInternational Journal of Cancer, 2000
- Pancreatic acinar carcinoma shows a distinct pattern of chromosomal imbalances by comparative genomic hybridizationGenes, Chromosomes and Cancer, 2000
- Fine Needle Aspiration Cytology of Acinar Cell Carcinoma of the PancreasActa Cytologica, 1996
- Only wild‐type c‐ki‐ras codons 12, 13, and 61 in human pancreatic acinar cell carcinomasMolecular Carcinogenesis, 1994
- Needle aspiration cytology of acinar‐cell carcinoma of the pancreas: Report of a case with diagnostic pitfalls and unusual ultrastructural findingsDiagnostic Cytopathology, 1994
- Acinar Cell Carcinoma of the PancreasThe American Journal of Surgical Pathology, 1992
- Rapid acinar to ductal transdifferentiation in cultured human exocrine pancreasThe Journal of Pathology, 1992
- Pancreatic carcinoma with polyarthritis, fat necrosis, and high serum lipase and trypsin activity.Gut, 1990