ROLE OF INSULIN AND COUNTER-REGULATORY HORMONES ON THE METABOLIC DETERIORATION THAT FOLLOWS INSULIN WITHDRAWAL IN DIABETIC-PATIENTS
- 1 March 1989
- journal article
- research article
- Vol. 45 (1) , 53-59
Abstract
In eight insulin dependent diabetic patients treated by continuous subcutaneous insulin infusion (1.1 .+-. 0.2 U/h), the levels (measured hourly from 23 h to 05 h) of blood glucose, non esterified fatty acids (NEFA), glycerol and 3-OH-butyrate (3-OH-B) have been correlated to the circulating levels of free insulin (FIRI), glucagon, growth hormone or cortisol, in two experimental conditions: A Insulin being infused as usual (physiological FIRI levels) and B, progressively declining FIRI levels (insulin infusion arrested at 23 h). In condition A, blood glucose levels correlated significantly to both insulin and glucagon; NEFA, glycerol and 3OH-B correlated only to insulin. In condition B, blood glucose was significantly correlated to insulin but not to glucagon while NEFA, glycerol and 3-OH-B were significantly correlated to both hormones but not to growth hormone or cortisol. Therefore, on the metabolic deterioration that follows insulin withdrawal, growth hormone and cortisol seem to play a minor role, the main role being played by the decrease in circulating insulin levels and to a lesser extent by the increase in glucagon levels.This publication has 11 references indexed in Scilit:
- Insulin-Stimulated Glucose Disposal Is not Increased in Anorexia Nervosa*Journal of Clinical Endocrinology & Metabolism, 1985
- A 6-hour nocturnal interruption of a continuous subcutaneous insulin infusion: 2. Marked attenuation of the metabolic deterioration by somatostatinDiabetologia, 1983
- A 6-hour nocturnal interruption of a continuous subcutaneous insulin infusion: 1. Metabolic and hormonal consequences and scheme for a prompt return to adequate controlDiabetologia, 1983
- Circadian profiles of blood glucose and plasma free insulin during treatment with Semisynthetic and Biosynthetic human insulin, and comparison with conventional monocomponent preparationsEuropean Journal of Clinical Pharmacology, 1983
- Cortisol-Induced Insulin Resistance in Man: Impaired Suppression of Glucose Production and Stimulation of Glucose Utilization due to a Postreceptor Defect of Insulin Action*Journal of Clinical Endocrinology & Metabolism, 1982
- Dose-response characteristics for effects of insulin on production and utilization of glucose in manAmerican Journal of Physiology-Endocrinology and Metabolism, 1981
- Altered Responsiveness to Cortisol, Epinephrine, and Glucagon in Insulin-infused Juvenile-onset Diabetics: A Mechanism for Diabetic InstabilityDiabetes, 1980
- The Temporal Relationship between Endogenously Secreted Stress Hormones and Metabolic Decompensation in Diabetic Man*Journal of Clinical Endocrinology & Metabolism, 1980
- Radioimmunoassay of corticosterone, cortisol and cortisone: Their application to human cord and maternal plasmaThe Journal of Steroid Biochemistry and Molecular Biology, 1978
- Microdetermination of Long-chain Fatty Acids in Plasma and TissuesJournal of Biological Chemistry, 1960