Permanent cell cycle exit in G2 phase after DNA damage in normal human fibroblasts
- 1 August 2003
- journal article
- Published by Springer Nature in The EMBO Journal
- Vol. 22 (15) , 3992-4002
- https://doi.org/10.1093/emboj/cdg387
Abstract
EMBO Press is an editorially independent publishing platform for the development of EMBO scientific publications.Keywords
This publication has 36 references indexed in Scilit:
- Induction of the Cdk inhibitor p21 by LY83583 inhibits tumor cell proliferation in a p53-independent mannerJournal of Clinical Investigation, 2002
- Multiple centrosomes arise from tetraploidy checkpoint failure and mitotic centrosome clusters in p53 and RB pocket protein-compromised cellsProceedings of the National Academy of Sciences, 2002
- E2F integrates cell cycle progression with DNA repair, replication, and G2/M checkpointsGenes & Development, 2002
- The Human Papillomavirus E6 protein and its contribution to malignant progressionOncogene, 2001
- Biological activities and molecular targets of the human papillomavirus E7 oncoproteinOncogene, 2001
- Neither p21WAF1 nor 14-3-3sigma prevents G2 progression to mitotic catastrophe in human colon carcinoma cells after DNA damage, but p21WAF1 induces stable G1 arrest in resulting tetraploid cells.2001
- Cell cycle checkpoint signaling through the ATM and ATR kinasesGenes & Development, 2001
- Tetraploid State Induces p53-dependent Arrest of Nontransformed Mammalian Cells in G1Molecular Biology of the Cell, 2001
- The HPV-16 E6 and E6-AP complex functions as a ubiquitin-protein ligase in the ubiquitination of p53Cell, 1993
- p53 is required for radiation-induced apoptosis in mouse thymocytesNature, 1993