Episodic activation off the coagulation system in unstable angina does not elicit an acute phase reaction

This publication has 10 references indexed in Scilit:

• Frequent sampling by clear venipuncture in unstable angina is a reliable method to assess haemostatic system activity
  Fibrinolysis, 1994
• The Prognostic Value of C-Reactive Protein and Serum Amyloid A Protein in Severe Unstable Angina
  New England Journal of Medicine, 1994
• Site of intimal rupture or erosion of thrombosed coronary atherosclerotic plaques is characterized by an inflammatory process irrespective of the dominant plaque morphology.
  Circulation, 1994
• Increased expression of neutrophil and monocyte adhesion molecules in unstable coronary artery disease.
  Circulation, 1993
• C-reactive protein increases production of IL-1α, IL-1β, and TNF-α, and expression of mRNA by human alveolar macrophages
  Journal of Leukocyte Biology, 1993
• Transient intermittent lymphocyte activation is responsible for the instability of angina.
  Circulation, 1992
• The Pathogenesis of Coronary Artery Disease and the Acute Coronary Syndromes
  New England Journal of Medicine, 1992
• Increased urinary leukotriene excretion in patients with cardiac ischemia. In vivo evidence for 5-lipoxygenase activation.
  Circulation, 1992
• Elevation of C-reactive protein in “active” coronary artery disease
  The American Journal of Cardiology, 1990
• Interleukin 1 acts on cultured human vascular endothelium to increase the adhesion of polymorphonuclear leukocytes, monocytes, and related leukocyte cell lines.
  Journal of Clinical Investigation, 1985