Two N-Terminal Self-Association Domains Are Required for the Dominant Negative Transcriptional Activity of WT1 Denys-Drash Mutant Proteins

This publication has 20 references indexed in Scilit:

• Truncated WT1 mutants alter the subnuclear localization of the wild-type protein.
  Proceedings of the National Academy of Sciences, 1995
• Antagonism of WT1 activity by protein self-association.
  Proceedings of the National Academy of Sciences, 1995
• WT1-mediated Transcriptional Activation Is Inhibited by Dominant Negative Mutant Proteins
  Journal of Biological Chemistry, 1995
• WT-1 is required for early kidney development
  Cell, 1993
• Alternative splicing and genomic structure of the Wilms tumor gene WT1.
  Proceedings of the National Academy of Sciences, 1991
• Germline mutations in the Wilms' tumor suppressor gene are associated with abnormal urogenital development in Denys-Drash syndrome
  Cell, 1991
• Clinicopathologic review of twelve children with nephropathy, Wilms tumor, and genital abnormalities (Drash syndrome)
  The Journal of Pediatrics, 1990
• Homozygous deletion in Wilms tumours of a zinc-finger gene identified by chromosome jumping
  Nature, 1990
• Isolation and characterization of a zinc finger polypeptide gene at the human chromosome 11 Wilms' tumor locus
  Cell, 1990
• A syndrome of pseudohermaphroditism, Wilms' tumor, hypertension, and degenerative renal disease
  The Journal of Pediatrics, 1970