Endogenous DNA abasic sites cause cell death in the absence of Apn1, Apn2 and Rad1/Rad10 in Saccharomyces cerevisiae
- 3 June 2002
- journal article
- Published by Springer Nature in The EMBO Journal
- Vol. 21 (11) , 2833-2841
- https://doi.org/10.1093/emboj/21.11.2833
Abstract
EMBO Press is an editorially independent publishing platform for the development of EMBO scientific publications.Keywords
This publication has 42 references indexed in Scilit:
- Identification of APN2, the Saccharomyces cerevisiae homolog of the major human AP endonuclease HAP1, and its role in the repair of abasic sitesGenes & Development, 1998
- Synergism between yeast nucleotide and base excision repair pathways in the protection against DNA methylation damageCurrent Genetics, 1998
- CDC5 and CKII Control Adaptation to the Yeast DNA Damage CheckpointPublished by Elsevier ,1997
- DNA glycosylases in the base excision repair of DNABiochemical Journal, 1997
- Inactivation of OGG1 increases the incidence of G · C→T · A transversions in Saccharomyces cerevisiae : evidence for endogenous oxidative damage to DNA in eukaryotic cellsMolecular Genetics and Genomics, 1997
- When Checkpoints FailCell, 1997
- Oxidative damage to DNA: Formation, measurement, and biological significancePublished by Springer Nature ,1997
- Requirement of mismatch repair genes MSH2 and MSH3 in the RAD1-RAD10 pathway of mitotic recombination in Saccharomyces cerevisiae.1996
- Single-Stranded DNA Arising at Telomeres in cdc13 Mutants May Constitute a Specific Signal for the RAD9 CheckpointMolecular and Cellular Biology, 1995
- Role of the Rad1 and Rad10 Proteins in Nucleotide Excision Repair and RecombinationJournal of Biological Chemistry, 1995