Genetic alterations in gliomas

This publication has 10 references indexed in Scilit:

• CDKN2 (p16/MTS1) gene deletion or CDK4 amplification occurs in the majority of glioblastomas.
  1994
• CDK4 amplification is an alternative mechanism to p16 gene homozygous deletion in glioma cell lines.
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• Molecular genetic analysis of oligodendroglial tumors shows preferential allelic deletions on 19q and 1p.
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• Amplification of multiple genes from chromosomal region 12q13-14 in human malignant gliomas: preliminary mapping of the amplicons shows preferential involvement of CDK4, SAS, and MDM2.
  1994
• Functional characterization of an EGF receptor with a truncated extracellular domain expressed in glioblastomas with EGFR gene amplification.
  1994
• A common region of homozygous deletion in malignant human gliomas lies between the IFN alpha/omega gene cluster and the D9S171 locus.
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• Gene and chromosomal alterations associated with the development of human gliomas
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• Amplification and overexpression of the MDM2 gene in a subset of human malignant gliomas without p53 mutations.
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• Histological Typing of Tumours of the Central Nervous System
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• Identical splicing of aberrant epidermal growth factor receptor transcripts from amplified rearranged genes in human glioblastomas.
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