Lipidic Pore Formation by the Concerted Action of Proapoptotic BAX and tBID
Open Access
- 1 July 2004
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 279 (29) , 30081-30091
- https://doi.org/10.1074/jbc.m313420200
Abstract
No abstract availableKeywords
This publication has 74 references indexed in Scilit:
- Bid, but Not Bax, Regulates VDAC ChannelsJournal of Biological Chemistry, 2004
- Fatty acids enhance membrane permeabilization by pro-apoptotic BaxBiochemical Journal, 2004
- Cardiolipin Is Not Required for Bax-mediated Cytochrome c Release from Yeast MitochondriaJournal of Biological Chemistry, 2004
- Bax-type Apoptotic Proteins Porate Pure Lipid Bilayers through a Mechanism Sensitive to Intrinsic Monolayer CurvatureJournal of Biological Chemistry, 2002
- The Apoptotic Protein tBid Promotes Leakage by Altering Membrane CurvatureJournal of Biological Chemistry, 2002
- Direct addition of BimL to mitochondria does not lead to cytochrome c releaseFEBS Letters, 2002
- Mitochondrial Lipid Alterations during Fas- and Radiation-Induced ApoptosisBiochemical and Biophysical Research Communications, 2001
- Proapoptotic BAX and BAK: A Requisite Gateway to Mitochondrial Dysfunction and DeathScience, 2001
- The Destabilization of Lipid Membranes Induced by the C-terminal Fragment of Caspase 8-cleaved Bid Is Inhibited by the N-terminal FragmentJournal of Biological Chemistry, 2000
- Biomembrane fusion: a new concept derived from model studies using two interacting planar lipid bilayersBiochimica et Biophysica Acta (BBA) - Reviews on Biomembranes, 1987