Genomic Imbalances in Pediatric Intracranial Ependymomas Define Clinically Relevant Groups
- 1 December 2002
- journal article
- research article
- Published by Elsevier in The American Journal of Pathology
- Vol. 161 (6) , 2133-2141
- https://doi.org/10.1016/s0002-9440(10)64491-4
Abstract
No abstract availableKeywords
This publication has 26 references indexed in Scilit:
- Gain of 1q and loss of 22 are the most common changes detected by comparative genomic hybridisation in paediatric ependymomaGenes, Chromosomes and Cancer, 2001
- Childhood cancer survival in EuropeEuropean Journal Of Cancer, 2001
- Gain of 1q Is Associated with Adverse Outcome in Favorable Histology Wilms’ TumorsThe American Journal of Pathology, 2001
- Multicentre analysis of patterns of DNA gains and losses in 204 neuroblastoma tumors: How many genetic subgroups are there?Medical and Pediatric Oncology, 2001
- Deletions below 10 megabasepairs are detected in comparative genomic hybridization by standard reference intervalsGenes, Chromosomes and Cancer, 1999
- A Multi-Institutional Retrospective Study of Intracranial Ependymoma in ChildrenJournal of Pediatric Hematology/Oncology, 1999
- Cytogenetics and prognosis in childhood lymphoblastic leukaemia: results of MRC UKALL XBritish Journal of Haematology, 1997
- Gain of chromosome arm 17q predicts unfavourable outcome in neuroblastoma patientsEuropean Journal Of Cancer, 1997
- Intracranial Ependymomas of ChildhoodNeurosurgery, 1995
- Treatment of intracranial ependymomas of children: review of a 15-year experienceInternational Journal of Radiation Oncology*Biology*Physics, 1994