A role for histone deacetylase HDAC1 in modulating the transcriptional activity of MyoD: inhibition of the myogenic program
- 2 April 2001
- journal article
- Published by Springer Nature in The EMBO Journal
- Vol. 20 (7) , 1739-1753
- https://doi.org/10.1093/emboj/20.7.1739
Abstract
EMBO Press is an editorially independent publishing platform for the development of EMBO scientific publications.Keywords
This publication has 76 references indexed in Scilit:
- Cyclin E–Cdk2 Phosphorylation Promotes Late G1-Phase Degradation of MyoD in Muscle CellsExperimental Cell Research, 2000
- HDAC4 deacetylase associates with and represses the MEF2 transcription factorThe EMBO Journal, 1999
- DNA damage activates p53 through a phosphorylation–acetylation cascadeGenes & Development, 1998
- Butyrate Stimulates Cyclin D and p21 and Inhibits Cyclin-Dependent Kinase 2 Expression in HT-29 Colonic Epithelial CellsBiochemical and Biophysical Research Communications, 1997
- Interdependence between muscle differentiation and cell-cycle controlBiochimica et Biophysica Acta (BBA) - Reviews on Cancer, 1997
- The Transcriptional Coactivators p300 and CBP Are Histone AcetyltransferasesPublished by Elsevier ,1996
- HLH forced dimers: Tethering MyoD to E47 generates a dominant positive myogenic factor insulated from negative regulation by IdCell, 1993
- Interplay between proliferation and differentiation within the myogenic lineageDevelopmental Biology, 1992
- 5-Bromo-2′-Deoxyuridine Blocks Myogenesis by Extinguishing Expression of MyoD1Science, 1989
- Expression of a single transfected cDNA converts fibroblasts to myoblastsPublished by Elsevier ,1987