Hypoxia-induced increase in soluble Flt-1 production correlates with enhanced oxidative stress in trophoblast cells from the human placenta
- 1 July 2004
- Vol. 26 (2-3) , 210-217
- https://doi.org/10.1016/j.placenta.2004.05.004
Abstract
No abstract availableKeywords
This publication has 27 references indexed in Scilit:
- Circulating Angiogenic Factors and the Risk of PreeclampsiaNew England Journal of Medicine, 2004
- Neutralization of Circulating Vascular Endothelial Growth Factor (VEGF) by Anti-VEGF Antibodies and Soluble VEGF Receptor 1 (sFlt-1) Induces ProteinuriaJournal of Biological Chemistry, 2003
- Vascular endothelial growth factor but not placental growth factor promotes trophoblast syncytialization in vitroJournal of the Society for Gynecologic Investigation, 2001
- Signal Transduction and Biological Function of Placenta Growth Factor in Primary Human Trophoblast1Biology of Reproduction, 1999
- Increased Circulating Levels of Vascular Endothelial Growth Factor in PreeclampsiaHypertension in Pregnancy, 1998
- Differential Transcriptional Regulation of the Two Vascular Endothelial Growth Factor Receptor GenesJournal of Biological Chemistry, 1997
- Hypoxia alters early gestation human cytotrophoblast differentiation/invasion in vitro and models the placental defects that occur in preeclampsia.Journal of Clinical Investigation, 1996
- Localisation of Placenta Growth Factor (PlGF) in Human Term PlacentaGrowth Factors, 1996
- Improved analysis of malondialdehyde in human body fluidsFree Radical Biology & Medicine, 1996
- Implantation and the placenta: key pieces of the development puzzleScience, 1994