Homodimeric and Heterodimeric Aryl Sulfotransferases Catalyze the Sulfuric Acid Esterification of N-Hydroxy-2-acetylaminofluorene

This publication has 28 references indexed in Scilit:

• Checkpoints of dueling dimers foil death wishes
  Cell, 1994
• Foreword
  Chemico-Biological Interactions, 1994
• Evidence of two separate mechanisms for the decrease in aryl sulfotransferase activity in rat liver during early stages of 2‐acetylaminofluorene–induced hepatocarcinogenesis
  Molecular Carcinogenesis, 1994
• Metabolic Activation ofN-Hydroxylated Metabolites of Carcinogenic and Mutagenic Arylamines and Arylamides by Esterification
  Drug Metabolism Reviews, 1994
• Sex-differentiated and growth-hormone-regulated mitoinhibition in rat liver during treatment with 2-acetylaminofluorene and partial hepatectomy in the resistant hepatocyte model
  Carcinogenesis: Integrative Cancer Research, 1991
• Myc meets its max
  Cell, 1991
• Self-catalyzed irreversible inactivation of rat hepatic aryl sulfotransferase IV by Nhydroxy-2-acetylaminofluorene
  Carcinogenesis: Integrative Cancer Research, 1990
• N-Sulfoöxy-2-aminofluorene is the major ultimate electrophilic and carcinogenic metabolite of N-hydroxy-2-acetylaminofluorene in the livers of infant male C57BL/6J × C3H/HeJ F₁ (B6C3F₁) mice
  Carcinogenesis: Integrative Cancer Research, 1985
• Role of sulfation in the formation of DNA adducts from N-hydroxy-2-acetylaminofluorene in rat liver in vivo. Inhibition of N-acetylated aminofluorene adduct formation by penta-chlorophenol
  Carcinogenesis: Integrative Cancer Research, 1981
• Identification of the persistently bound form of the carcinogen N-acetyl-2-aminofluorene to rat liver DNA in vivo
  Chemico-Biological Interactions, 1976