Abstract
Quantitative ECG and accessory circulatory and respiratory mass spectrographic studies were carried out on 30 young, healthy human subjects at rest during moderately acute, standardized stepwise exposure to simulated altitude up to 7000 m [PAO2 (partial pressure of O2) 26.9 mmHg]. The ECG (Einthoven and Wilson leads) and respiratory gases were recorded synchronously during ascent and descent. The extensive data assembled, summarized in 6 tables and illustrated in 7 graphs, represent the basis for future investigations on the single factors which underlie hypoxia-induced ECG changes during exposure to high altitude.

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